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We read the article by Shim and colleagues describing homotypic antibody-dependent-enhancement (ADE) of Zika virus (ZIKV) infections by low-level ZIKV-specific antibodies (1) with great interest. ADE predominantly describes a mechanism by which infectious antibody-virus immune complexes efficiently bind to Fc gamma receptors of immune cells, facilitating the infection of otherwise poorly susceptible cells. As noted by Shim et al., ADE has been described for genetically diverse viruses, including feline coronaviruses, human immunodeficiency virus type 1, and cytomegalovirus (2, 3). Although likely not solely …

Complex Scenario of Homotypic and Heterotypic Zika Virus Immune Enhancement