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The ability ofEscherichia coli K to support bacteriophage T4rII replication starts to decline at 3 to 6 min after infection by λ. This inhibition appears to depend on expression of the λrex + gene, since little inhibition was observed following infection by a λrex mutant or by a hybrid bacteriophage λi434 which lacks a functionalrex gene. For promotion of the synthesis ofrex product, cII+ and N+ genes are required and can acttrans , whereas cY+ , also required, must becis to arex + gene. These genes presumably play a role in the transcription of the cI-rex operon because they are also known to be required for repressor (cI product) synthesis. Functional cIII, O, P genes are not necessary for amplerex product synthesis. We also observed fullrex expression after infection by λsus cI mutants, suggesting thatrex and repressor are separate gene products and that repressor is not required for inhibition of T4rII replication. We also report experiments with arex mutant that is not leaky when in a lysogen but is sufficiently leaky shortly after infection to cause inhibition of T4rII replication.

Regulation of λ rex Expression After Infection of Escherichia coli K by Lambda Bacteriophage