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Osmotic stress was found to induce biofilm formation in a Staphylococcus aureus mucosal isolate. Inactivation of a global regulator of the bacterial stress response, the alternative transcription factor sigma(B), resulted in a biofilm-negative phenotype and loss of salt-induced biofilm production. Complementation of the mutant strain with an expression plasmid encoding sigma(B) completely restored the wild-type phenotype. The combined data suggest a critical role of sigma(B) in S. aureus biofilm regulation under environmental stress conditions.

Alternative Transcription Factor ς B Is Involved in Regulation of Biofilm Expression in a Staphylococcus aureus Mucosal Isolate

Alternative Transcription Factor ς ^B Is Involved in Regulation of Biofilm Expression in a Staphylococcus aureus Mucosal Isolate