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We have observed that human neutrophils (polymorphonuclear leukocytes [PMNs]) have an increased growth-inhibitory and killing effect on a strain of Candida albicans with a deletion of CHK1, a gene encoding a putative histidine kinase. The PMN effect was not due to increased phagocytosis of the null strain. This observation may partially explain the reduced virulence in a hematogenously disseminated murine model of candidiasis.

infection and immunity

Deletion of the Two-Component Histidine Kinase Gene ( <i>CHK1</i> ) of <i>Candida albicans</i> Contributes to Enhanced Growth Inhibition and Killing by Human Neutrophils In Vitro

Deletion of the Two-Component Histidine Kinase Gene ( CHK1 ) of Candida albicans Contributes to Enhanced Growth Inhibition and Killing by Human Neutrophils In Vitro