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Since interleukin-6 (IL-6) may promote Th2 responses, we infected BALB IL-6-deficient (IL-6(-/-)) mice with Leishmania major. There was not a significant difference between the courses of infection (lesion size and parasite burden) in IL-6(-/-) and wild-type mice, but IL-6(-/-) mice expressed lower levels of Th2- and Th1-associated cytokines.

infection and immunity

Interleukin-6 Deficiency Influences Cytokine Expression in Susceptible BALB Mice Infected with<i>Leishmania major</i>but Does Not Alter the Outcome of Disease

Interleukin-6 Deficiency Influences Cytokine Expression in Susceptible BALB Mice Infected withLeishmania majorbut Does Not Alter the Outcome of Disease