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Use of specific fimbria-defective mutants derived from Actinomyces viscosus T14 has shown that salivary acidic proline-rich protein 1 and statherin serve as receptors for type 1 fimbriae which mediate attachment of the organism to experimental pellicles on apatitic surfaces.

Adsorbed salivary proline-rich protein 1 and statherin: receptors for type 1 fimbriae of Actinomyces viscosus T14V-J1 on apatitic surfaces