English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

Intracerebral inoculation of 4-day-old rats with lymphocytic choriomeningitis virus (E-350 strain) produced a nonfatal, acute, severe, and permanent cerebellar necrosis with minimal histological evidence of inflammation. Virus persisted in the brain at high titers for 30 to 40 days and was finally cleared about 120 days after infection. Rabbit anti-rat lymphoid serum prevented cerebellar necrosis, and brain virus titers were higher than in control animals receiving normal rabbit serum. Thus, the immune response which clearly is responsible for the cerebellar lesion also plays a role in the gradual suppression of infection.

infection and immunity

Pathogenesis of Cerebellar Hypoplasia Produced by Lymphocytic Choriomeningitis Virus Infection of Neonatal Rats: Protective Effect of Immunosuppression with Anti-Lymphoid Serum