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Two viruses of the herpes group, herpes simplex virus and pseudorabies virus, cause an increase in the activity of thymidine kinase (E.C. 2.7.1.21) in baby hamster kidney cells (BHK 21) and primary rabbit kidney cells respectively (Klemperer et al. 1967; Hamada, Kamiya & Kaplan, 1966). Klemperer et al. (1967) showed that the thymidine kinase produced in cells infected with herpes simplex virus was inhibited by antiserum prepared against RK 13 cells (Beale, Christofinis & Furminger, 1963) infected with the same virus. Similar results were obtained by Hamada et al. (1966) using thymidine kinase induced by pseudorabies virus and antiserum prepared against cells infected with pseudorabies virus.Hamada et al. (1966) suggested that the increased activity and altered serological properties of thymidine kinase might have been the result of allosteric alterations in the enzyme caused by environmental changes within the infected cell.

The Immunological Specificity of Thymidine Kinases in Cells Infected by Viruses of the Herpes Group