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In utero retinoid exposure results in numerous craniofacial malformations, including craniosynostosis. Although many malformations associated with retinoic acid syndrome are associated with neural crest defects, the specific mechanisms of retinoid-induced craniosynostosis remain unclear. The authors used the culture of mouse cranial suture-derived mesenchymal cells to probe the potential cellular mechanisms of this teratogen to better elucidate mechanisms of retinoid-induced suture fusion.

Retinoic Acid Enhances Osteogenesis in Cranial Suture–Derived Mesenchymal Cells: Potential Mechanisms of Retinoid-Induced Craniosynostosis