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Sexual dimorphism in the degree of high blood pressure (BP) has been observed in both animal and human hypertension. However, the mechanisms are still poorly understood. We tested the hypothesis that long-term loss of sex steroids promotes changes in mesenteric vascular reactivity that impact the maintenance of hypertension in the spontaneously hypertensive rats (SHR).

Blood Pressure and Mesenteric Vascular Reactivity in Spontaneously Hypertensive Rats 7 Months After Gonadectomy