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The role of tumor necrosis factor-α (TNF-α), Janus kinase/signal transducers and activators of transcription (JAK/STAT) pathway, and mitochondrial Permeability Transition Pore in atorvastatin-induced cardioprotection were examined in human myocardium, in vitro.

anesthesiology

Atorvastatin-induced Cardioprotection of Human Myocardium Is Mediated by the Inhibition of Mitochondrial Permeability Transition Pore Opening <i>via</i> Tumor Necrosis Factor-α and Janus Kinase/Signal Transducers and Activators of Transcription Pathway

Atorvastatin-induced Cardioprotection of Human Myocardium Is Mediated by the Inhibition of Mitochondrial Permeability Transition Pore Opening via Tumor Necrosis Factor-α and Janus Kinase/Signal Transducers and Activators of Transcription Pathway