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PURPOSE: Chronic wounds are among the most common complications of diabetes mellitus with 5–10% of diabetics suffering from foot ulcers. Physiologic wound healing involves the complex interplay of multiple cellular components, including endothelial cells as a major angiogenic workhorse. We have previously shown that the absence of effective Nrf2 signaling contributes to pathologic diabetic wounds. The purpose of this study is to analyze the detrimental effects to endothelial cell function that result from a relative loss of Nrf2 within in vitro and in vivo models.

Abstract 119: Designing a Murine Model of the Foreign Body Reaction