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The effect of the glutamate analogue L- α-aminoadipate ( αAA) on the release of glutamate and γ-aminobutyric acid (GABA) from rat hippocampal slices was investigated in vitro. Oxygen/glucose deprivation caused a large release of glutamate and GABA. αAA added during energy deprivation reduced the glutamate release in a dose-dependent manner (56% reduction at 5 m M), whereas GABA release was unchanged. We speculate that ischemic glutamate release from the brain is mediated by a low affinity transport mechanism that is blocked by αAA.

L-α-Aminoadipate Reduces Glutamate Release from Brain Tissue Exposed to Combined Oxygen and Glucose Deprivation