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Obstructive sleep apnea (OSA) has been implicated in the pathophysiology of metabolic syndrome. Its contribution to insulin resistance is complicated by obesity and puberty. We hypothesized that OSA is associated with worse insulin resistance and lower adiponectin after adjustment for obesity and puberty and that catecholamines might mediate these changes.

Catecholamines, Adiponectin, and Insulin Resistance as Measured by HOMA in Children with Obstructive Sleep Apnea