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In Sjögren's syndrome (SS), salivary acinar cells are destroyed even though ductal cells are frequently spared from destruction and can sometimes proliferate. We made the hypothesis that abnormalities of the tumour suppressor protein p53, either by mutations leading to proliferation or by activation of the functional wild-type p53, explain this phenomenon.

A new defensive mechanism to prevent apoptosis in salivary ductal cells from patients with Sjögren's syndrome: over‐expression of p53 and p21