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In statins, the risk of muscle injury has been related to genetic variations and drug interactions. There is an increased risk of muscle toxicity in patients with SLCO1B1 polymorphism. Elevated CPKs are found in drug-drug interactions with medications that are strong inhibitors of the CYP3A4. The leading mechanism on statin induced myopathy is thought to be secondary to abnormal mitochondrial function with depletion of CoQ10 leading to decreased protein synthesis which may lead to myopathy.

A Multisite Review of Concomitant Use of Daptomycin and Statins and the Effect on CPK