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Treatment of astrocytoma is frequently hampered by radioresistance of the tumor. In addition to overexpression of ErbB1/EGFR, functional crosstalk between receptor tyrosine kinases and cell adhesion molecules may also contribute to therapy resistance.

neuro-oncology

Molecular interactions of ErbB1 (EGFR) and integrin- 1 in astrocytoma frozen sections predict clinical outcome and correlate with Akt-mediated in vitro radioresistance