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standard methods. RESULTS: CAVI level was 8.2260.18 m/sec in diabetic CKD patients and significantly higher than non-diabetic CKD (7.6160.18 m/sec) and controls (7.5960.17 m/sec). FGF-23 level was higher in CKD groups than controls, but not statistically significant. MCP-1 level was significantly higher in diabetic CKD patients. Klotho and sclerostin levels were signicantly lower in diabetic CKD patients. In CKD patients, CAVI showed positive correlations with age (r1⁄40.447 p<0.0001), cigarette smoking (r1⁄40.331, p1⁄40.035), mean arterial pressure (r1⁄40.327, p<0.0001), fasting blood glucose (r1⁄40.185, p1⁄40.033), HbA1c (r1⁄40.258, p1⁄4 0.003). Stepwise regression analysis revealed that age (p1⁄40.0001, B1⁄40.461), mean arterial blood pressure ( p<0.0001, B1⁄40.365), HbA1c (p1⁄40.003, B1⁄40.251) and MCP-1 (p1⁄40.013, B1⁄40.214) independently predicted CAVI levels.

FP332ESTIMATED PREVALENCE OF CHRONIC KIDNEY DISEASE AND ITS RISK FACTORS IN NORTH-CENTRAL NIGERIA: ANALYSIS OF AGGREGATE DATA FROM EIGHT COMMUNITIES