Serum hepcidin-25 levels in patients with chronic kidney disease are independent of glomerular filtration rate | Zendy

Hilde P.E. Peters | Zendy; Coby M. Laarakkers | Zendy; D. W. Swinkels | Zendy; Jack F.M. Wetzels | Zendy

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Serum hepcidin-25 levels in patients with chronic kidney disease are independent of glomerular filtration rate

Author(s) -

Hilde P.E. Peters,

Coby M. Laarakkers,

D. W. Swinkels,

Jack F.M. Wetzels

Publication year - 2009

Publication title -

nephrology dialysis transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.654

H-Index - 168

eISSN - 1460-2385

pISSN - 0931-0509

DOI - 10.1093/ndt/gfp546

Subject(s) - hepcidin , medicine , kidney disease , renal function , endocrinology , homeostasis , regulator , iron homeostasis , kidney , metabolism , anemia , biochemistry , biology , gene

Hepcidin is a key regulator of iron homeostasis and levels are elevated in patients with chronic kidney disease (CKD). Hepcidin may explain the often observed imbalance in iron metabolism in patients with CKD. We evaluated the influence of estimated glomerular filtration rate (eGFR) on serum levels of hepcidin-25 and its isoforms in patients with renal dysfunction.

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