Renal accumulation of pentosidine in non-diabetic proteinuria-induced renal damage in rats | Zendy

Femke Waanders | Zendy; Wendela L Greven | Zendy; John Baynes | Zendy; Suzanne R. Thorpe | Zendy; Andrea B. Kramer | Zendy; Ryoji Nagai | Zendy; Noriyuki Sakata | Zendy; Harry van Goor | Zendy; Gerjan Navis | Zendy

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Renal accumulation of pentosidine in non-diabetic proteinuria-induced renal damage in rats

Author(s) -

Femke Waanders,

Wendela L Greven,

John Baynes,

Suzanne R. Thorpe,

Andrea B. Kramer,

Ryoji Nagai,

Noriyuki Sakata,

Harry van Goor,

Gerjan Navis

Publication year - 2005

Publication title -

nephrology dialysis transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.654

H-Index - 168

eISSN - 1460-2385

pISSN - 0931-0509

DOI - 10.1093/ndt/gfh939

Subject(s) - medicine , pentosidine , proteinuria , glycation , pathogenesis , diabetic nephropathy , diabetes mellitus , advanced glycation end product , urology , endocrinology , glomerulopathy , glomerulosclerosis , kidney

Advanced glycation end-products (AGEs) contribute to the pathogenesis of diabetic glomerulopathy. The role of AGEs in non-diabetic renal damage is not well characterized. First, we studied whether renal AGE accumulation occurs in non-diabetic proteinuria-induced renal damage and whether this is ameliorated by renoprotective treatment. Secondly, we investigated whether renal AGE accumulation was due to intrarenal effects of local protein trafficking.

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