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Numerous studies over the past 15 years in diVerent cellular calcium in the pathophysiology of ischaemic injury models and cell types have demonstrated an acute renal failure (ARF ) in vivo. Chemically dissimilar increase in cytosolic calcium in renal epithelial cell calcium channel blockers (CCB) are eVective in pre- injury. Some of these studies are summarized in venting or attenuating the course of experimental isch- Table 1. However, despite these studies, crucial ques- aemic ARF (4). Vascular perturbations in experimental tions to implicate calcium as the primary factor in cell ischaemic ARF were found to be associated with an injury have remained. Does the increase in calcium increase in calcium in aVerent arterioles from ischaemic

Calcium-mediated proximal tubular injury—what is the role of cysteine proteases?