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Plague is initiated by Yersinia pestis, a highly virulent bacterial pathogen. In late stages of the infection, bacteria proliferate extensively in the internal organs despite the massive infiltration of neutrophils. The ineffective inflammatory response associated with tissue damage may contribute to the low efficacy of antiplague therapies during late stages of the infection. In the present study, we address the possibility of improving therapeutic efficacy by combining corticosteroid administration with antibody therapy in the mouse model of bubonic plague.

the journal of infectious diseases (online. university of chicago press)/the journal of infectious diseases

Adjunctive Corticosteroid Treatment Against<i>Yersinia pestis</i>Improves Bacterial Clearance, Immunopathology, and Survival in the Mouse Model of Bubonic Plague

Adjunctive Corticosteroid Treatment AgainstYersinia pestisImproves Bacterial Clearance, Immunopathology, and Survival in the Mouse Model of Bubonic Plague