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vs. 31±4 years), pregnancy weeks (30.9±5.7 vs. 30.4±4.3), primi-/multiparity status, and heart rate (90±11 vs. 85±10 bpm). GHT/PE group had a blood pressure (BP) of 157±24/100±30 mmHg, whereas control group had a BP of 109±3/66±11 mmHg. GHT/PE group had decreased global systolic function, as assessed by 4D LVEF, by comparison with the control group: 53.2±5.7% vs. 57.7%±6.0%, p=0.007. Meanwhile, GHT/PE group had decreased regional systolic function: S wave (10.4±2.4 vs. 12.7±2.3 cm/s), radial strain (29.7±8.0 vs. 47.4±15.0) and strain rate (2.1±0.4 vs. 3.1±0.4), global longitudinal strain (-17.7±2.7 vs. 21.1±2.0) and strain rate (-1.2±0.2 vs. 1.3±0.1) (all p<0.001); but with a nonsignificant decrease in circumferential strain and strain rate. No significant differences in any parameters of vascular function were found between groups. Conclusions: Patients with gestational hypertension or preeclampsia had subclinical cardiac systolic dysfunction. Further studies should establish the subtle mechanisms, and whether this condition is transitory or it persists after discharge, increasing the risk for progression to heart failure in these patients.

P3257The impact of maternal hemodynamics on preterm delivery and intrauterine growth restriction in gestational hypertension