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Crohn's disease [CD] is a complex disorder characterised by an inappropriate immune response, impaired barrier function and microbial dysbiosis. Mutations in nucleotide oligomeriation domain 2 [NOD2] are CD risk factors. Increase of intestinal permeability, CD4 + T cell infiltration, and bacterial dysbiosis are also seen in Nod2-knockout [Nod2 KO ] mice. However, the specificity and relationship between these Nod2-associated abnormalities remain largely unexplored.

Nod2Deficiency Leads to a Specific and Transmissible Mucosa-associated Microbial Dysbiosis Which Is Independent of the Mucosal Barrier Defect