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The therapeutic potential of Hedgehog (Hh) signalling agonists for vascular diseases is of growing interest. However, molecular and cellular mechanisms underlying the role of the Hh signalling in vascular biology remain poorly understood. The purpose of the present article is to clarify some conflicting literature data.

Full-length Dhh and N-terminal Shh act as competitive antagonists to regulate angiogenesis and vascular permeability