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This study demonstrates the role of BM-MSCs’ microvesicles on MM cells’ phenotype and translation initiation status (TI). MVs from MM patients’ BM-MSCs elevate MM cells’ TI and consequently their proliferation, migration, invasion, and autophagy whereas normal donors’ BM-MSCs MVs do not.

carcinogenesis

Microvesicles derived from normal and multiple myeloma bone marrow mesenchymal stem cells differentially modulate myeloma cells’ phenotype and translation initiation