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This study reports the role of RIPK1 in modulating HNSCC metastasis. We find RIPK1 is downregulated during the tumor progression and reduced RIPK1 expression correlates with promoter hypermethylation. In tumor-derived HNSCC cell lines, RIPK1 silencing enhances the tumor promoting properties while augmenting the apoptotic response to synthetic dsRNA.

Innate immune signaling through differential RIPK1 expression promote tumor progression in head and neck squamous cell carcinoma