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Tobacco smoking is an apparent cofactor with HPVs in cervical malignancies, but the interactions are poorly understood. We modeled tobacco smoke exposure to cervical epithelium; data suggest that tobacco smoke contributes to early stages of HPV-related cancer progression.

carcinogenesis

Tobacco exposure results in increased E6 and E7 oncogene expression, DNA damage and mutation rates in cells maintaining episomal human papillomavirus 16 genomes