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Actin perturbation by latrunculins (Lat) reduces recruitment of base excision repair factors to laser-induced damage, while nocodazole treatment increases it. Surprisingly, LatA leads to the nuclear accumulation of both actin and tubulin. Since actin perturbation aggravates Zeocin-induced cell death, this links cytoskeletal integrity to base excision repair.

Cytoskeleton integrity influences XRCC1 and PCNA dynamics at DNA damage