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Acquired hearing loss is a worldwide epidemic that affects all ages. It is multifactorial in etiology with poorly characterized molecular mechanisms. Mitochondria are critical components in hearing. Here, we aimed to identify the mechanisms of mitochondria-dependent hearing loss using Fus1 KO mice, our novel model of mitochondrial dysfunction/oxidative stress.

antioxidants and redox signaling

Novel Role of the Mitochondrial Protein Fus1 in Protection from Premature Hearing Loss<i>via</i>Regulation of Oxidative Stress and Nutrient and Energy Sensing Pathways in the Inner Ear

Novel Role of the Mitochondrial Protein Fus1 in Protection from Premature Hearing LossviaRegulation of Oxidative Stress and Nutrient and Energy Sensing Pathways in the Inner Ear