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Systemic lupus erythematosus (SLE) patients' peripheral blood lymphocytes (PBL) show mitochondrial dysfunction and oxidative stress. To determine the electrochemical bases of mitochondrial dysfunction, we measured electron transport chain (ETC) activity and its regulation by N-acetylcysteine (NAC) that reversed glutathione depletion and improved disease activity in SLE. ETC activity was assessed in PBL of 69 SLE patients and 37 healthy donors. Negatively isolated T cells were examined in 7 SLE patients, 11 healthy donors, and 10 nonlupus inflammatory arthritis (IA) donors.

antioxidants and redox signaling

Increased Mitochondrial Electron Transport Chain Activity at Complex I Is Regulated by<i>N</i>-Acetylcysteine in Lymphocytes of Patients with Systemic Lupus Erythematosus

Increased Mitochondrial Electron Transport Chain Activity at Complex I Is Regulated byN-Acetylcysteine in Lymphocytes of Patients with Systemic Lupus Erythematosus