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Yersinia pestis, the causative agent of plague, showed a temperature-dependent change in lipid A composition, with a reduced degree of acylation when bacteria were grown at 37 degrees C (tetraacylated) versus ambient temperature (hexaacylated).

Tetraacylated Lipopolysaccharide ofYersinia pestisCan Inhibit Multiple Toll‐Like Receptor–Mediated Signaling Pathways in Human Dendritic Cells