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Understanding the immunologic mechanisms that can control the progression of HIV disease is paramount for the development of vaccines or of new strategies for immunotherapy. Whether infection occurs perinatally, sexually, or parenterally, disease ultimately develops in all but a very few individuals, although the time to disease is quite variable. This tremendous variability in disease progression may be influenced by age at the time of infection, viral load, race, and host genetic factors, most notably HLA class and CCR5 [1–8]; however, the influence of gender is more controversial. Additionally, there is evidence that these same factors may also protect against infection in some individuals and in certain settings.

The Role of Race and Gender in T Cell Responses in Children Perinatally Infected with HIV‐1