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In cardiac myocytes, electrical signaling and Ca2+ signaling are closely coupled. Electrical signals, in the form of action potentials, produce periodic increases and decreases in intracellular [Ca2+]—so-called “Ca2+ transients”—and these Ca2+ signals can, in turn, influence action

A privileged role for neuronal Na+ channels in regulating ventricular [Ca2+] and arrhythmias