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The ClC-K2 chloride channel has long been known to robustly express in the basolateral plasma membranes of epithelial cells lining the thick ascending limb, distal convoluted tubule, and distal nephron (including the collecting duct system; [Stauber et al., 2012][1]; [Jentsch, 2015][2]). Our

Alkaline activation of ClC-K2 chloride channels switches renal cells from reabsorbing to secreting