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(From the Laboratory of Physiology ~n the Harvard Med/~ca~ ~vhool.) In the experiments recorded in this paper, the heart of the cat was kept beating by passing defibrinated cat's blood from the aorta through the coronary vessels into the right auricle and ventricle, whence it escaped through a cannula in the pulmonary artery. The blood thus transfused through the coronary vessels into the right heart flowed from the tube in the pulmonary artery in drops, as a rule. The volume of the transfused blood was measured by counting the drops. The volume could be increased or diminished at will by increasing or diminishing the pressure of the blood at the mouths of the coronary arteries. A ttiirthle membrane manometer, connected with the left ventricle by a tube passed through the left auricular appendix and mitral valve, measured the force and frequency of ventricu]ar contraction , the left ventricle and the connecting tubes being filled with normal saline solution. The experiments were performed in a warm chamber at a sufficiently constant temperature. The results of the investigation show that the force of ventricular contraction is immediately affected by a change in the amount of blood supplied to the cardiac muscle, the ventricle beating with more force if the volume of the coronary circulation is increased, and with less force if it is diminished; and this holds true within wide, but, as yet, not accurately determined limits. The frequency of ventricular contraction, on the contrary, is largely independent of the blood supply to the cardiac muscle, and may remain almost unchanged

ON THE RELATION OF THE VOLUME OF THE CORONARY CIRCULATION TO THE FREQUENCY AND FORCE OF THE VENTRICULAR CONTRACTION IN THE ISOLATED HEART OF THE CAT