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erythematosus arising in a patient with chronic silicosis. may be formed and released from macrophages. Parallels to Rheumatol Int 1997; 16: 217–218. the pathogenesis of silica-induced scleroderma are given, as 7. Siebels M, Schulz V, Andrassy K. Systemischer Lupus erythobserved in 111 patients in our department (14). ematodes und Silikose. Dtsch Med Wochenschr 1995; 120: As silica cannot be removed from the body, its precipitating 214–218. effect cannot be stopped. This indicates that the clinical course 8. Silicosis and Silicate Disease Committee. Diseases associated with is that of classical autoimmune diseases, with progressions and exposure to silica and nonfibrous silicate minerals. Arch Pathol remissions. The therapy has to be in line with these facts and Lab Med 1988; 112: 673–720. has to be adjusted according to the criteria of the clinical 9. Doll JN, Stankus RP, Hughes J, Weill H, Gupta RC, Rodriguez activity of the disease. The best way to prevent this type of M, et al. Immune complexes and autoantibodies in silicosis. J Allergy 1981; 68: 281–285. SLE is to minimize the exposure to silica. 10. Jones RN, Turner-Warwick M, Ziskind M, Weill H. High prevalence of antinuclear antibodies in sandblaster’s silicosis. Am Rev Respir Dis 1976; 113: 393–395. REFERENCES 11. Frank R, Giese T, Dummer R, Walther T, Rytter M, Ziegler V, et al. Silica-induced cytokine release in human monocyte cultures 1. Kilburn KH, Warshaw RH. Prevalence of symptoms of systemic and its possible involvement in the pathophysiology of silicalupus erythematosus (SLE) and of fluorescent antinuclear antiassociated scleroderma. Eur J Dermatol 1993; 3: 304–309. bodies associated with chronic exposure to trichlorethylene and 12. Anderegg U, Vorberg S, Herrmann K, Haustein U-F. Silica other chemicals in well water. Environmental Res 1992; 57: 1–9. directly induces intercellular adhesion molecule 1 (ICAM-1) 2. Hatron PY, Plouvier B, Francois M. Assoziation von systemisexpression in cultured endothelial cells. Eur J Dermatol 1997; chem Lupus erythematodes und Silikose. Kasuistik (5 Pat.), Rev 7: 27–31. Med Interne 1982; 3: 245–246, ref. in: Zbl Haut-u Geschl-Krkh 13. Anderegg U, Vorberg S, Herrmann K, Haustein U-F. Increased 1983/84; 149(1): 403. expression of interstitial collagenase in silica-treated fibroblasts. 3. Ebihara I, Kawami M. Health hazards of mineral dust with Eur J Dermatol 1996; 6/1: 51–55. special reference to the causation of immuno-pathologic systemic 14. Haustein U-F, Herrmann K. Environmental scleroderma. Clin diseases. J Science Labour 1985; 61: 1–31. Dermatol 1994; 12: 467–473. 4. Mehlhorn J, Gerlach Ch. Gemeinsames Auftreten von Silikose und Lupus erythematodes. Z Erkr Atm-Org 1990; 175: 38–41. Accepted July 24, 1997. 5. Ziegler V, Pfeil B, Haustein U-F. Berufliche Quarzstaubexposition – Progressive Sklerodermie und Lupus erythematodes. Z Hautkr H+G 1991; 66: 968–970. Uwe-Frithjof Haustein, Department of Dermatology, University of Leipzig, Liebigstr. 21, DE-04103 Leipzig, Germany. 6. Özoran K, Uzan H, Tutkak H, Caner N, Yücel M. Systemic lupus

Centroblastic-centrocytic lymphoma arising at the site of previous herpes zoster eruption