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Significance Sickle cell trait has repeatedly been identified as a major human malaria resistance factor. Despite this, the exact mechanism of resistance is unclear. These studies demonstrate how the evolutionarily significant sickle hemoglobin affectsPlasmodium falciparum infection success and leads to a better understanding of the molecular basis and pathogenesis of malaria infection.

Resistance to Plasmodium falciparum in sickle cell trait erythrocytes is driven by oxygen-dependent growth inhibition