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life (>90%), whereas healthy adults are unlikely to become chronically infected (<5%). In many developing countries, HBV is transmitted from chronically infected mothers to their babies, who in turn become chronically infected, perpetuating the cycle of transmission.Ithadbeenproposedthattheabilityofthevirustopersistin the newborn and infants is due to an immune tolerance caused by the secreted hepatitis B ‘e’ antigen (HBeAg), which can cross the placenta. Recent work has confirmed that HBeAg can modulate the innate immune pathway by suppression of Toll-like receptors 2 . Diagnosis of HBV infection relies on interpretation of serological assays designed to detect viral antigens and their corresponding antibodies. The most useful marker is serum hepatitis B surface antigen (HBsAg), a non-infectious mosaic of the viral surface glycoproteins that circulate in the blood in great excess during the course of infection. This was the so-called Australia antigen, originally discovered by Blumberg and colleagues in the blood of an AboriginalAustralianwheninvestigatingimmunologicalvariationof complement proteins 3 . It was some time before the association of

Chronic viral hepatitis and hepatitis B virus infection: stop the cancer