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m/s n° 6-7, vol. 33, juin-juillet 2017 DOI : 10.1051/medsci/20173306016 IscR in gene regulation and DNA recognition. Biochim Biophys Acta 2015 ; 1854 : 1101-12. 7. Teixido L, Carrasco B, Alonso JC, et al. Fur activates the expression of Salmonella enterica pathogenicity island 1 by directly interacting with the hilD operator in vivo and in vitro. PLoS One 2011 ; 6 : e19711. 8. Miller HK, Kwuan L, Schwiesow L, et al. IscR is essential for yersinia pseudotuberculosis type III secretion and virulence. PLoS Pathog 2014 ; 10 : e1004194. 9. Golinelli-Cohen MP, Bouton C. Fe-S proteins acting as redox switch: new key actor of cellular adaptative responses. Curr Chem Biol 2017 (sous presse). 10. Choby JE, Mike LA, Mashruwala AA, et al. A smallmolecule inhibitor of iron-sulfur cluster assembly uncovers a link between virulence regulation and metabolism in staphylococcus aureus. Cell Chem Biol 2016 ; 23 : 1351-61. 2. Ellermeier JR, Slauch JM. Adaptation to the host environment: regulation of the SPI1 type III secretion system in Salmonella enterica serovar Typhimurium. Curr Opin Microbiol 2007 ; 10 : 24-9. 3. Py B, Barras F. Du fer et du soufre dans les protéines. Comment la cellule construit-elle les cofacteurs fer-soufre essentiels à son fonctionnement ? Med Sci (Paris) 2014 ; 30 : 1110-22. 4. Velayudhan J, Karlinsey JE, Frawley ER, et al. Distinct roles of the Salmonella enterica serovar Typhimurium CyaY and YggX proteins in the biosynthesis and repair of iron-sulfur clusters. Infect Immun 2014 ; 82 : 1390-401. 5. Vergnes A, Viala JP, Ouadah-Tsabet R, et al. The ironsulfur cluster sensor IscR is a negative regulator of Spi1 type III secretion system in Salmonella enterica. Cell Microbiol 2016 ; 19 : doi: 10.1111/cmi.12680. 6. Santos JA, Pereira PJ, Macedo-Ribeiro S. What a difference a cluster makes: The multifaceted roles of bactériennes comme récemment démontré pour S. aureus [10]. ‡ Control of the Salmonella enterica virulence by iron-sulfur cluster biogenesis machinery

Les lésions hépatiques induites par la cholestase : le rôle de S1PR2