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Hypocotyl elongation is influenced by light and hormones, but the molecular mechanisms underlying this process are not yet fully elucidated. We had previously suggested that the Arabidopsis DOF transcription factor DAG1 may be a negative component of the mechanism of light-mediated inhibition of hypocotyl elongation, as light-grown  dag1  knock-out mutant seedlings show significant shorter hypocotyls than the wild type. By using high-throughput RNA-seq, we compared the transcriptome profile of  dag1  and wild type hypocotyls and seedlings. We identified more than 250 genes differentially expressed in  dag1  hypocotyls, and their analysis suggests that DAG1 is involved in the promotion of hypocotyl elongation through the control of ABA, ethylene and auxin signaling. Consistently, ChIP-qPCR results show that DAG1 directly binds to the promoters of  WRKY18  encoding a transcription factor involved in ABA signaling, of the ethylene- induced gene  ETHYLENE RESPONSE FACTOR  ( ERF2 ), and of the  SMALL AUXIN UP RNA 67  ( SAUR67 ), an auxin-responding gene encoding a protein promoting hypocotyl cell expansion.

Genome-wide RNA-seq analysis indicates that the DAG1 transcription factor promotes hypocotyl elongation acting on ABA, ethylene and auxin signaling