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The COP9 signalosome (CSN) is an evolutionary conserved multiprotein complex that regulates many aspects of plant development by controlling the activity of CULLIN-RING E3 ubiquitin ligases (CRLs). CRLs ubiquitinate and target for proteasomal degradation a vast number of specific substrate proteins involved in many developmental and physiological processes, including light and hormone signaling and cell division. As a consequence of CSN pleiotropic function, complete loss of CSN activity results in seedling lethality. Therefore, a detailed analysis of CSN physiological functions in adult Arabidopsis plants has been hampered by the early seedling lethality of  csn  null mutants. Here we report the identification and characterization of a viable allele of the Arabidopsis COP9 signalosome subunit 4 ( CSN4 ). The allele, designated  csn4-2035 , suppresses the adventitious root (AR) phenotype of the Arabidopsis  superroot2-1  mutant, potentially by altering its auxin signaling. Furthermore, we show that although the  csn4-2035  mutation affects primary and lateral root (LR) formation in the  2035  suppressor mutant,  CSN4  and other subunits of the  COP9  complex seem to differentially control AR and LR development.

The Arabidopsis Cop9 signalosome subunit 4 (CSN4) is involved in adventitious root formation