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Many genomic alterations associated to human diseases localize in non-coding regulatory elements located far from the promoters they regulate, making the association of non-coding mutations or risk associated variants to target genes challenging. The range of action of a given set *Corresponding authors: fcasfer@upo.es; jlgomska@upo.es. #These authors contribute equally to this work ACCESSION CODES: GSE27945; GSE27944 AUTHOR CONTRIBUTIONS JLGS and FC conceived the study, designed the experiments, interpreted results and wrote the manuscript. DM devised bioinformatics methods, performed data analysis and wrote the paper. CP, MS, MAB, conducted the mouse ChIP experiments. CVQ, MFM and FRT performed the chicken ChIP experiments. EdCM, EM and LM performed the insulator assays. AFM conducted the 3C experiments. FM, AA and NF provided the PBMCs from blood cells and performed the qRT-PCR, CNRA/CNRB activity on a luciferase reported system, quantification of GFI1 relative expression of 108 PBMC samples, genotyping of the EVI5 rs11804321 and statistical analysis OD performed the high-throughput sequencing. OB, LT, IO and PSP performed data analysis. MM and RG collaborated in the experimental design, discussion of results and in writing the manuscript. NIH Public Access

Erratum: Genome-wide CTCF distribution in vertebrates defines equivalent sites that aid the identification of disease-associated genes