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In the January 2006 issue, Chan et al.1 reported a significant association between severe acute respiratory syndrome (SARS) and a variable number of tandem repeats (VNTR) polymorphism in exon 4 of CLEC4M in a collection of individuals from Hong Kong. CLEC4M encodes L-SIGN ('liver/lymph node-specific ICAM-3 grabbing nonintegrin'), which serves as a receptor for many viruses, including SARS coronavirus (CoV)2.

Lack of support for an association between CLEC4M homozygosity and protection against SARS coronavirus infection