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Phytophthora  pathogens secrete an array of specific effector proteins to manipulate host innate immunity to promote pathogen colonization. However, little is known about the host targets of effectors and the specific mechanisms by which effectors increase susceptibility. Here we report that the soybean pathogen  Phytophthora sojae  uses an essential effector PsAvh262 to stabilize endoplasmic reticulum (ER)-luminal binding immunoglobulin proteins (BiPs), which act as negative regulators of plant resistance to  Phytophthora . By stabilizing BiPs, PsAvh262 suppresses ER stress-triggered cell death and facilitates  Phytophthora  infection. The direct targeting of ER stress regulators may represent a common mechanism of host manipulation by microbes.

A Phytophthora sojae effector suppresses endoplasmic reticulum stress-mediated immunity by stabilizing plant Binding immunoglobulin Proteins