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In stunned myocardium oxygen consumption is relatively high compared with the reduced ventricular function. On the other hand, inotropic stimulation is frequently required to improve postischemic ventricular dysfunction. However, inotropic agents which act via intracellular increased calcium result in a higher oxygen demand. Therefore Ca(2+)-sensitization might be a favorable alternative.

Myocardial efficiency in stunned myocardium. Comparison of Ca2+-sensitization and PDE III-inhibition on energy consumption✩