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An increase in stimulation frequency can facilitate or depress cardiac Ca2+ current (ICa). The aim was to examine the Ca2+ dependence of these effects, to determine if facilitation is sustained, and to elucidate the mechanism by which isoprenaline modulates facilitation.

Use-dependent facilitation and depression of ?-type Ca2+ current in guinea-pig ventricular myocytes: modulation by Ca2+ and isoprenaline