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High-density lipoprotein (HDL) is believed to protect against development of atherosclerosis by inhibiting the accumulation of oxidized lipids in low-density lipoprotein (LDL). Paradoxically, HDL lipid is more susceptible to oxidation than LDL lipid. In the present study, we examined the effect of oxidized phospholipids on the uptake of HDL by macrophages.

Formation of apolipoprotein AI–phosphatidylcholine core aldehyde Schiff base adducts promotes uptake by THP-1 macrophages