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Stress proteins (heat shock proteins, HSPs) are molecular chaperones that have been shown to enhance the survival of cells exposed to environmental stress. We sought to investigate the effects of hypoxia on the levels of HSP27 and heme oxygenase-1 (HO-1 or HSP32) in an established model of rat neonatal cardiac myocytes in culture.

cardiovascular research

Hypoxia differentially regulates stress proteins in cultured cardiomyocytes Role of the p38 stress-activated kinase signaling cascade, and relation to cytoprotection