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Hypothesis / aims of study The time-course study shows that bladder dysfunction following bladder outlet obstruction (BOO) changes from compensation (hypersensitive bladder, HSB), detrusor overactivity (DO) to decompensation (detrusor underactive, DU). Previous studies have shown increased urothelial cell apoptosis and suburothelial inflammation in patients with bladder storage problems, such as interstitial cystitis, ketamine-related cystitis and recurrent urinary tract infection. The aim of this study was to investigate the urothelial inflammation and urothelial cell apoptosis in patients with bladder outlet obstruction with different bladder dysfunction.

Urothelial dysfunction and chronic inflammation in patients with bladder outlet obstruction and different bladder dysfunction and different BOO degree